Browsing by Yonsei Author : Um, Ji Won

Name:
엄지원 [Um, Ji Won]
Department :
College of Medicine - Dept. of Physiology

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Issue DateTitleYonsei Author(s)Type
2017Synaptic functions of the IQSEC family of ADP-ribosylation factor guanine nucleotide exchange factors엄지원Article
2016IQ Motif and SEC7 Domain-containing Protein 3 (IQSEC3) Interacts with Gephyrin to Promote Inhibitory Synapse Formation엄지원Article
2016SALM5 trans-synaptically interacts with LAR-RPTPs in a splicing-dependent manner to regulate synapse development. 엄지원Article
2016Emergent Synapse Organizers: LAR-RPTPs and Their Companions엄지원Article
2016LRRTM3 Regulates Excitatory Synapse Development through Alternative Splicing and Neurexin Binding 엄지원Article
2016Metabotropic glutamate receptor 5 couples cellular prion protein to intracellular signalling in Alzheimer's disease.엄지원Article
2016Neurotrophin-3 Regulates Synapse Development by Modulating TrkC-PTPσ Synaptic Adhesion and Intracellular Signaling Pathways.엄지원Article
2016SALM4 suppresses excitatory synapse development by cis-inhibiting trans-synaptic SALM3-LAR adhesion 엄지원Article
2016Slitrk Missense Mutations Associated with Neuropsychiatric Disorders Distinctively Impair Slitrk Trafficking and Synapse Formation 엄지원, 이영호Article
2016Proteolytic degradation and potential role of onconeural protein cdr2 in neurodegeneration엄지원Article
2015The balancing act of GABAergic synapse organizers엄지원Article
2015PTPσ functions as a presynaptic receptor for the glypican-4/LRRTM4 complex and is essential for excitatory synaptic transmission 엄지원Article
2014Structural basis for LAR-RPTP/Slitrk complex-mediated synaptic adhesion엄지원Article
2014Calsyntenins function as synaptogenic adhesion molecules in concert with neurexins 엄지원Article
2014Elfn1 recruits presynaptic mGluR7 in trans and its loss results in seizures엄지원Article
2013LAR-RPTPs: synaptic adhesion molecules that shape synapse development엄지원Article
2013Metabotropic glutamate receptor 5 is a coreceptor for Alzheimer aβ oligomer bound to cellular prion protein 엄지원Article
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