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YUHSpace1. College of Medicine (의과대학)Yonsei Biomedical Research Center (연세의생명연구원)1. Journal Papers

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Showing results 8 to 22 of 22

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Issue DateTitleJournal Title
2015Histone methylation levels correlate with TGFBIp and extracellular matrix gene expression in normal and granular corneal dystrophy type 2 corneal fibroblasts BMC MEDICAL GENOMICS
2011Inhibition of TGFBIp expression by lithium: implications for TGFBI-linked corneal dystrophy therapy INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE
2016Inhibition of TGFBIp expression reduces lymphangiogenesis and tumor metastasisONCOGENE
2015Inhibitory Effect of Tranilast on Transforming Growth Factor-Beta-Induced Protein in Granular Corneal Dystrophy Type 2 Corneal FibroblastsCORNEA
2010Involvement of TGF-{beta} receptor- and integrin-mediated signaling pathways in the pathogenesis of granular corneal dystrophy II INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE
2016Lithium inhibits tumor lymphangiogenesis and metastasis through the inhibition of TGFBIp expression in cancer cells SCIENTIFIC REPORTS
2020Lysosomal dysfunction of corneal fibroblasts underlies the pathogenesis of Granular Corneal Dystrophy Type 2 and can be rescued by TFEB JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
2015Lysosomal trafficking of TGFBIp via caveolae-mediated endocytosis PLOS ONE
2013Melatonin induces autophagy via an mTOR-dependent pathway and enhances clearance of mutant-TGFBIpJOURNAL OF PINEAL RESEARCH
2011Melatonin protects against oxidative stress in granular corneal dystrophy type 2 corneal fibroblasts by mechanisms that involve membrane melatonin receptorsJOURNAL OF PINEAL RESEARCH
2017Melatonin reduces endoplasmic reticulum stress and corneal dystrophy-associated TGFBIp through activation of endoplasmic reticulum-associated protein degradationJOURNAL OF PINEAL RESEARCH
2016Pathogenesis and treatments of TGFBI corneal dystrophiesPROGRESS IN RETINAL AND EYE RESEARCH
2017Role of TGFBIp in Wound Healing and Mucin Expression in Corneal Epithelial Cells YONSEI MEDICAL JOURNAL
2016TGF-β regulates TGFBIp expression in corneal fibroblasts via miR-21, miR-181a, and Smad signalingBIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
2020Uptake of cell debris and enhanced expression of inflammatory factors in response to dead cells in corneal fibroblast cellsEXPERIMENTAL EYE RESEARCH
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