Hemintaxia-hypesthesia in thalamic stroke has been rarely reported. We experienced six patients who has hemiataxia-hypesthesia with or without transientweakness due to thalamic stroke. Upon reviewing the pattern of sensory deficits and the presence of weakness m our cases and those in previously reported thalamic stroke patients with hemiataxiahypesthesia with or without transient weakness, hemiataxia-hypesthesia has a localizing value of the thalamic lesion, particularly in case of presenting with pain sensory loss and of no weakness.