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Submicroscopic deletion of FGFR1 gene is recurrently detected in myeloid and lymphoid neoplasms associated with ZMYM2-FGFR1 rearrangements: a case study

DC Field Value Language
dc.contributor.author박서진-
dc.contributor.author최종락-
dc.date.accessioned2014-12-19T16:21:02Z-
dc.date.available2014-12-19T16:21:02Z-
dc.date.issued2012-
dc.identifier.issn0001-5792-
dc.identifier.urihttps://ir.ymlib.yonsei.ac.kr/handle/22282913/89362-
dc.description.statementOfResponsibilityopen-
dc.relation.isPartOfACTA HAEMATOLOGICA-
dc.rightsCC BY-NC-ND 2.0 KR-
dc.rights.urihttps://creativecommons.org/licenses/by-nc-nd/2.0/kr/-
dc.subject.MESHBase Sequence-
dc.subject.MESHChromosome Deletion-
dc.subject.MESHChromosomes, Human, Pair 8/genetics-
dc.subject.MESHDNA-Binding Proteins/genetics*-
dc.subject.MESHFatal Outcome-
dc.subject.MESHGene Deletion*-
dc.subject.MESHGene Rearrangement*-
dc.subject.MESHHumans-
dc.subject.MESHMale-
dc.subject.MESHMiddle Aged-
dc.subject.MESHMolecular Sequence Data-
dc.subject.MESHMyeloproliferative Disorders/genetics*-
dc.subject.MESHReceptor, Fibroblast Growth Factor, Type 1/genetics*-
dc.subject.MESHTranscription Factors/genetics*-
dc.titleSubmicroscopic deletion of FGFR1 gene is recurrently detected in myeloid and lymphoid neoplasms associated with ZMYM2-FGFR1 rearrangements: a case study-
dc.typeArticle-
dc.contributor.collegeCollege of Medicine (의과대학)-
dc.contributor.departmentDept. of Laboratory Medicine (진단검사의학)-
dc.contributor.googleauthorYang J.J.-
dc.contributor.googleauthorPark T.S.-
dc.contributor.googleauthorChoi J.R.-
dc.contributor.googleauthorPark S.-J.-
dc.contributor.googleauthorCho S.Y.-
dc.contributor.googleauthorJun K.R.-
dc.contributor.googleauthorKim H.R.-
dc.contributor.googleauthorLee J.N.-
dc.contributor.googleauthorOh S.H.-
dc.contributor.googleauthorLee S.-
dc.contributor.googleauthorKim B.-
dc.contributor.googleauthorMarschalek R.-
dc.contributor.googleauthorMeyer C.-
dc.identifier.doi22236811-
dc.admin.authorfalse-
dc.admin.mappingfalse-
dc.contributor.localIdA01494-
dc.contributor.localIdA04182-
dc.relation.journalcodeJ00014-
dc.identifier.eissn1421-9662-
dc.identifier.pmid22236811-
dc.identifier.urlhttp://www.karger.com/Article/Abstract/334707-
dc.subject.keywordBase Sequence-
dc.subject.keywordChromosome Deletion-
dc.subject.keywordChromosomes, Human, Pair 8/genetics-
dc.subject.keywordDNA-Binding Proteins/genetics*-
dc.subject.keywordFatal Outcome-
dc.subject.keywordGene Deletion*-
dc.subject.keywordGene Rearrangement*-
dc.subject.keywordHumans-
dc.subject.keywordMale-
dc.subject.keywordMiddle Aged-
dc.subject.keywordMolecular Sequence Data-
dc.subject.keywordMyeloproliferative Disorders/genetics*-
dc.subject.keywordReceptor, Fibroblast Growth Factor, Type 1/genetics*-
dc.subject.keywordTranscription Factors/genetics*-
dc.contributor.alternativeNamePark, Seo Jin-
dc.contributor.alternativeNameChoi, Jong Rak-
dc.contributor.affiliatedAuthorPark, Seo Jin-
dc.contributor.affiliatedAuthorChoi, Jong Rak-
dc.citation.volume127-
dc.citation.number2-
dc.citation.startPage119-
dc.citation.endPage123-
dc.identifier.bibliographicCitationACTA HAEMATOLOGICA, Vol.127(2) : 119-123, 2012-
dc.identifier.rimsid34558-
dc.type.rimsART-
Appears in Collections:
1. College of Medicine (의과대학) > Dept. of Laboratory Medicine (진단검사의학교실) > 1. Journal Papers

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