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Alternative lengthening of telomeres is mechanistically linked to potential therapeutic vulnerability in the stem-like subtype of gastric cancer

DC Field Value Language
dc.contributor.author정재호-
dc.date.accessioned2022-03-11T05:49:14Z-
dc.date.available2022-03-11T05:49:14Z-
dc.date.issued2021-09-
dc.identifier.urihttps://ir.ymlib.yonsei.ac.kr/handle/22282913/187812-
dc.description.statementOfResponsibilityopen-
dc.languageEnglish-
dc.publisherWiley-
dc.relation.isPartOfCLINICAL AND TRANSLATIONAL MEDICINE-
dc.rightsCC BY-NC-ND 2.0 KR-
dc.subject.MESHDrug Resistance, Neoplasm / genetics-
dc.subject.MESHEnergy Metabolism / genetics-
dc.subject.MESHEpithelial-Mesenchymal Transition / genetics-
dc.subject.MESHHumans-
dc.subject.MESHProtein Interaction Maps / genetics-
dc.subject.MESHStomach Neoplasms* / genetics-
dc.subject.MESHStomach Neoplasms* / pathology-
dc.subject.MESHTelomere / genetics*-
dc.subject.MESHTelomere Homeostasis / genetics*-
dc.titleAlternative lengthening of telomeres is mechanistically linked to potential therapeutic vulnerability in the stem-like subtype of gastric cancer-
dc.typeArticle-
dc.contributor.collegeCollege of Medicine (의과대학)-
dc.contributor.departmentDept. of Surgery (외과학교실)-
dc.contributor.googleauthorJi-Yong Sung-
dc.contributor.googleauthorJae-Ho Cheong-
dc.identifier.doi10.1002/ctm2.561-
dc.contributor.localIdA03717-
dc.relation.journalcodeJ04026-
dc.identifier.eissn2001-1326-
dc.identifier.pmid34586731-
dc.contributor.alternativeNameCheong, Jae Ho-
dc.contributor.affiliatedAuthor정재호-
dc.citation.volume11-
dc.citation.number9-
dc.citation.startPagee561-
dc.identifier.bibliographicCitationCLINICAL AND TRANSLATIONAL MEDICINE, Vol.11(9) : e561, 2021-09-
Appears in Collections:
1. College of Medicine (의과대학) > Dept. of Surgery (외과학교실) > 1. Journal Papers
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