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Current concepts in the immunopathogenesis of psoriasis

DC FieldValueLanguage
dc.contributor.author유욱-
dc.date.accessioned2020-09-04T02:12:25Z-
dc.date.available2020-09-04T02:12:25Z-
dc.date.issued2004-10-
dc.identifier.issn0733-8635-
dc.identifier.urihttps://ir.ymlib.yonsei.ac.kr/handle/22282913/178871-
dc.description.abstractThere is much evidence to support the concept that psoriasis is a type 1 autoimmune disease, primarily mediated by interferon gamma and other inflammatory cytokines. There has been renewed interest in the role of components of the innate immune system, however,and it may be that overlap between the innate and acquired arms of the immune system can better explain immunopathogenesis in psoriasis. Relevant cell types, receptors, and immune mediators within these traditional boundaries of the immune system are discussed.Finally, pathogenic contributions from important psoriatic mouse models and recent genomic data using the new gene chip technology are elaborated.-
dc.description.statementOfResponsibilityrestriction-
dc.languageEnglish-
dc.publisherElsevier Health Sciences Division-
dc.relation.isPartOfDERMATOLOGIC CLINICS-
dc.rightsCC BY-NC-ND 2.0 KR-
dc.subject.MESHAnimals-
dc.subject.MESHBiopsy, Needle-
dc.subject.MESHCytokines / immunology*-
dc.subject.MESHCytokines / metabolism-
dc.subject.MESHDisease Models, Animal-
dc.subject.MESHDisease Progression-
dc.subject.MESHFemale-
dc.subject.MESHHumans-
dc.subject.MESHImmunity, Innate / physiology*-
dc.subject.MESHImmunohistochemistry-
dc.subject.MESHMale-
dc.subject.MESHPrognosis-
dc.subject.MESHPsoriasis / immunology*-
dc.subject.MESHPsoriasis / pathology*-
dc.subject.MESHRats-
dc.subject.MESHSensitivity and Specificity-
dc.titleCurrent concepts in the immunopathogenesis of psoriasis-
dc.typeArticle-
dc.contributor.collegeCollege of Medicine (의과대학)-
dc.contributor.departmentDept. of Dermatology (피부과학교실)-
dc.contributor.googleauthorMichelle A Lowes-
dc.contributor.googleauthorWook Lew-
dc.contributor.googleauthorJames G Krueger-
dc.identifier.doi10.1016/j.det.2004.03.010-
dc.contributor.localIdA02487-
dc.relation.journalcodeJ00706-
dc.identifier.eissn1558-0520-
dc.identifier.pmid15450332-
dc.identifier.urlhttps://www.sciencedirect.com/science/article/abs/pii/S0733863504000191-
dc.contributor.alternativeNameLew, Wook-
dc.contributor.affiliatedAuthor유욱-
dc.citation.volume22-
dc.citation.number4-
dc.citation.startPage349-
dc.citation.endPage369-
dc.identifier.bibliographicCitationDERMATOLOGIC CLINICS, Vol.22(4) : 349-369, 2004-10-
Appears in Collections:
1. College of Medicine (의과대학) > Dept. of Dermatology (피부과학교실) > 1. Journal Papers

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