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YUHSpace1. College of Medicine (의과대학)BioMedical Science Institute (의생명과학부)1. Journal Papers

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Issue DateTitleJournal Title
2014Characterization of gene expression and activated signaling pathways in solid-pseudopapillary neoplasm of pancreasMODERN PATHOLOGY
2015Clinicopathologic Features and Molecular Characteristics of Glucose Metabolism Contributing to ¹⁸F-fluorodeoxyglucose Uptake in Gastrointestinal Stromal Tumors PLOS ONE
2017Clinicopathological characteristics of KIT and protein kinase C-δ expression in adenoid cystic carcinoma: comparison with chromophobe renal cell carcinoma and gastrointestinal stromal tumourHISTOPATHOLOGY
2012Genomic Grade Index predicts postoperative clinical outcome of GIST BRITISH JOURNAL OF CANCER
2014High expression of AT-rich interactive domain 3A (ARID3A) is associated with good prognosis in colorectal carcinomaANNALS OF SURGICAL ONCOLOGY
2013Identification and Selective Degradation of Neopeptide-Containing Truncated Mutant Proteins in the Tumors with High Microsatellite InstabilityCLINICAL CANCER RESEARCH
2011Identification of frequently mutated genes with relevance to nonsense mediated mRNA decay in the high microsatellite instability cancers.INTERNATIONAL JOURNAL OF CANCER
2017Identification of specifically activated angiogenic molecules in HMGB-1-induced angiogenesis BMB REPORTS
2017mRNAs containing NMD-competent premature termination codons are stabilized and translated under UPF1 depletion SCIENTIFIC REPORTS
2017Prognosis of stage III colorectal carcinomas with FOLFOX adjuvant chemotherapy can be predicted by molecular subtype ONCOTARGET
2016Promoter methylation of PCDH10 by HOTAIR regulates the progression of gastrointestinal stromal tumors ONCOTARGET
2018Survivin is a novel transcription regulator of KIT and is downregulated by miRNA-494 in gastrointestinal stromal tumors INTERNATIONAL JOURNAL OF CANCER
2017Sustained Mutant KIT Activation in the Golgi Complex Is Mediated by PKC-θ in Gastrointestinal Stromal TumorsCLINICAL CANCER RESEARCH
2019β-catenin activation down-regulates cell-cell junction-related genes and induces epithelial-to-mesenchymal transition in colorectal cancers SCIENTIFIC REPORTS
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