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Tumor necrosis factor-α signaling via TNFR1/p55 is deleterious whereas TNFR2/p75 signaling is protective in adult infarct myocardium.

DC FieldValueLanguage
dc.contributor.author윤영섭-
dc.date.accessioned2018-05-10T06:33:50Z-
dc.date.available2018-05-10T06:33:50Z-
dc.date.issued2011-
dc.identifier.issn0065-2598-
dc.identifier.urihttps://ir.ymlib.yonsei.ac.kr/handle/22282913/158208-
dc.description.statementOfResponsibilityopen-
dc.languageEnglish-
dc.publisherKluwer Academic/Plenum Publishers-
dc.relation.isPartOfAdvances in Experimental Medicine and Biology-
dc.rightsCC BY-NC-ND 2.0 KR-
dc.rightshttps://creativecommons.org/licenses/by-nc-nd/2.0/kr/-
dc.subject.MESHAdult-
dc.subject.MESHAging-
dc.subject.MESHAngiogenesis Inducing Agents/metabolism-
dc.subject.MESHAnimals-
dc.subject.MESHApoptosis-
dc.subject.MESHBlood Donors-
dc.subject.MESHCapillaries/metabolism-
dc.subject.MESHCapillaries/pathology-
dc.subject.MESHCardiotonic Agents/metabolism-
dc.subject.MESHEndothelial Cells/metabolism-
dc.subject.MESHEndothelial Cells/pathology-
dc.subject.MESHHumans-
dc.subject.MESHMice-
dc.subject.MESHMice, Knockout-
dc.subject.MESHMyocardial Infarction/complications-
dc.subject.MESHMyocardial Infarction/metabolism*-
dc.subject.MESHMyocardial Infarction/pathology-
dc.subject.MESHMyocardial Infarction/prevention & control*-
dc.subject.MESHMyocardial Ischemia/complications-
dc.subject.MESHMyocardial Ischemia/metabolism-
dc.subject.MESHMyocardial Ischemia/pathology-
dc.subject.MESHMyocardium/metabolism-
dc.subject.MESHMyocardium/pathology*-
dc.subject.MESHNeovascularization, Pathologic/metabolism-
dc.subject.MESHReceptors, Tumor Necrosis Factor, Type I/metabolism*-
dc.subject.MESHReceptors, Tumor Necrosis Factor, Type II/metabolism*-
dc.subject.MESHSignal Transduction*-
dc.subject.MESHStem Cells/metabolism-
dc.subject.MESHStem Cells/pathology-
dc.subject.MESHSurvival Analysis-
dc.subject.MESHTroponin I/metabolism-
dc.subject.MESHTumor Necrosis Factor-alpha/metabolism*-
dc.titleTumor necrosis factor-α signaling via TNFR1/p55 is deleterious whereas TNFR2/p75 signaling is protective in adult infarct myocardium.-
dc.typeArticle-
dc.contributor.collegeCollege of Medicine-
dc.contributor.departmentDept. of Life Science-
dc.contributor.googleauthorRaj Kishore-
dc.contributor.googleauthorTengiz Tkebuchava-
dc.contributor.googleauthorSharath P. Sasi-
dc.contributor.googleauthorMarcy Silver-
dc.contributor.googleauthorHu-Ya Gilbert-
dc.contributor.googleauthorYoung-Sup Yoon-
dc.contributor.googleauthorHee-Young Park-
dc.contributor.googleauthorTina Thorne-
dc.contributor.googleauthorDouglas W. Losordo-
dc.contributor.googleauthorDavid A. Goukassian-
dc.identifier.doi10.1007/978-1-4419-6612-4_45-
dc.contributor.localIdA02579-
dc.relation.journalcodeJ03108-
dc.identifier.eissn2214-8019-
dc.identifier.pmid21153348-
dc.contributor.alternativeNameYoon, Young Sup-
dc.contributor.affiliatedAuthorYoon, Young Sup-
dc.citation.volume691-
dc.citation.startPage433-
dc.citation.endPage448-
dc.identifier.bibliographicCitationAdvances in Experimental Medicine and Biology, Vol.691 : 433-448, 2011-
Appears in Collections:
1. College of Medicine (의과대학) > BioMedical Science Institute (의생명과학부) > 1. Journal Papers

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