Browsing by Yonsei Author : Kong, Kyoung Ah

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Name:
Kong, Kyoung Ah [공경아]
Department :
College of Medicine (의과대학) - Dept. of Anatomy (해부학교실)

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Showing results 1 to 13 of 13

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Issue DateTitleJournal Title
2014Akt1 mediates the posterior Hoxc gene expression through epigenetic modifications in mouse embryonic fibroblastsBIOCHIMICA ET BIOPHYSICA ACTA-GENE REGULATORY MECHANISMS
2014Pax3 function is required specifically for inner ear structures with melanogenic fatesBIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
2013Direct Interaction Between Akt1 and Gcn5 and its Plausible Function on Hox Gene Expression in Mouse Embryonic Fibroblast Cells Journal of Experimental & Biomedcal Science (대한의생명과학회지)
2013Akt1 as a putative regulator of Hox genesGENE
2012Post-transcriptional Regulation of Gcn5, a Putative Regulator of Hox in Mouse Embryonic Fibroblast Cells Journal of Experimental & Biomedcal Science (대한의생명과학회지)
2012A novel protein Jpk induces bacterial cell death through reactive oxygen species.GENE
2011Transient retinoic acid signaling confers anterior-posterior polarity to the inner ear PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
2009Clinical and molecular characterizations of novel POU3F4 mutations reveal that DFN3 is due to null function of POU3F4 protein. PHYSIOLOGICAL GENOMICS
2009The third helix of the Hoxc8 homeodomain peptide enhances the efficiency of gene transfer in combination with lipofectamine.MOLECULAR BIOTECHNOLOGY
2008The third helix of the murine Hoxc8 homeodomain facilitates protein transduction in mammalian cellsBIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
2007ER Stress Induces the Expression of Jpk, which Inhibits Cell Cycle Progression in F9 Teratocarcinoma CellANNALS OF THE NEW YORK ACADEMY OF SCIENCES
2005ER Stress-Induced Jpk Expression and the Concomitant Cell Death Journal of Experimental & Biomedcal Science (대한의생명과학회지)
2003A Novel Gene, Jpk, Induces Apoptosis in F9 Murine Teratocarcinoma Cell through ROS GenerationANNALS OF THE NEW YORK ACADEMY OF SCIENCES
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